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However, in 2001, a handful of groundbreaking research discoveries confirmed that the Ca 2+ ionophore ionomycin could trigger an apoptosis-like cell death in erythrocytes associated with the appearance of apoptotic signs, including cell shrinkage, membrane vesiculation, or PS exposure [22, 23]

Functional consequences of age-dependent changes in glutathione status in the brain

Permeability of bilayer lipid membranes for superoxide (O2) radicals