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Quercetin can inhibit oxidative damage induced by oxidized LDLoxldl in endothelial cells by activating SIRT1 and regulating AMPK signaling (Hung et al., 2015), and can also regulate the expression of various chromatin modifiers in a dose-dependent manner, such as reducing the activity of DNMT and HDACs and down-regulating global DNA methylation levels (Kedhari Sundaram et al., 2019)

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Chondrocyte affinity peptides can also increase the integration of scaffolds and tissues due to their ability to bind cells at the sites of defects

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