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However, l-serine supplementation does not reduce hyperglycemia in BKS- db/db models despite the improvement in neuropathy phenotypes, suggesting that some forms of diabetic peripheral neuropathy are more directly driven by l-serine deficiency as opposed to hyperglycemia

A commonly agreed upon the fact in the pathogenesis of SLE is the dysfunction of T cells, B cells, dendritic cells, macrophages, and neutrophils, likely triggered by genes and/or the environment [4]

Chimienti F, Devergnas S, Favier A, Seve M

Lets use a common example: a 1mg vial of IGF-1 LR3
Liu et al., 1996
