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This post-translational modification is now recognised as a major mechanism of redox-regulated cell signalling, controlling the activity of transcription factors, kinases, phosphatases, and metabolic enzymes in response to the intracellular redox environment

A study in human GBMs showed that NETs promote IL-8 secretion in GBMs by stimulating the NF-B signalling pathway, which in turn stimulates endothelial cells to generate blood vessels to deliver essential nutrients and oxygen to the tumour site (101)

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The tumor-specific isoform ct-OATP1B3-V1 enhances chemotherapy sensitivity by localizing to the plasma membrane and importing drugs