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Description
Addressing underlying medical conditions that contribute to vitamin deficiency is also crucial
Thus, GPx-1-deficient neurons have enhanced cell death after exposure to Abeta, whereas ebselen or N-acetyl-L-cysteine treatment or overexpression of GPx-1 significantly attenuated Abeta-induced toxicity (200)
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Inferring causal effects of homocysteine and B-vitamin concentrations on bone mineral density and fractures: Mendelian randomization analyses
Human half-life unknown
