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The mechanism may be related to curculigoside significantly reducing AD-promoting factors (e.g., A1-42, p-tau) and increasing ferroptosis protective factors (e.g., GPX4, SLC7A11, GSH) in the hippocampus and cortex of AD mice, 574 suggesting that the natural compound curculigoside can be used as a promising therapeutic agent to improve AD by inhibiting ferroptosis

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Cell 47 , 656663 (2012)

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Q., Liu, N

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In addition, the authors demonstrate that with supplementation of reduced GSH, there is improved intracellular control of an M

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