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As an endogenous phospholipid and a main component of surfactant, DPPC has a high surfactantpenetrating, lungtargeting capability and excellent biocompatibility [103]

These mice survived for a total of 25 days and naturally died, which we defined as the survival endpoint as illustrated in Fig

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CDK1 directly binds and phosphorylates the S447 site of ACSL4, promoting the ubiquitination degradation of ACSL4, while genetic and pharmacological blocking of CDK1 enhances ACSL4 activity, promoting ferroptosis of colon cancer cells and increasing their susceptibility to oxaliplatin

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