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If immunotherapy aims to establish protective immune memory comparable to that induced by acute infection, then its failure to do socoupled with the insufficient tumor-killing capacity that permits on-treatment progressionreveals fundamental limitations in current strategies and underscores the need for a mechanistic explanation

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This is clinically relevant because many supplementation strategies, including N-acetylcysteine (NAC) therapy, target this bottleneck

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4 Biological role of MTH1 in tumors Tumor cells, characterized by chronic overactivity in mitotic and pro-survival signaling, undergo metabolic changes that generate high levels of oxidants