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Lack of coupling between the NADP redox status and E GSH in glr1 cells, provides a powerful genetic system to separate NADP and E GSH -dependent responses
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(2020) knocked down the expression of DNMT1 and DNMT3a in primary cultured hippocampal neurons and found that hippocampal neurons exhibited fewer and thinner dendritic branches, lower density of excitatory synapse formation, and could inhibit Glu-induced Ca2+ influx
