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The p-chlorophenylalanine (PCPA) inhibits tryptophan hydroxylase, therefore acting as a serotonergic toxin (Jquier et al., 1967) (see Table 5)
While the current studies primarily focus on vascular endothelial dysfunction as central contributors to hypertension-induced ED, neural damage is also a significant factor
Subcellular and cellular locations of nitric-oxide synthase isoforms as determinants of health and disease
Lastly, EPR spectroscopy revealed no change in hydroxyl radical generation with copper addition
Research progress of radiation induced bystander and abscopal effects in normal tissue
