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Description
These results suggest a neuroprotective effect of GPX1 against the peroxynitrite-induced SGN damage

They concluded that GSH is the primary sulfur receptor of SQOR in vivo , and proposed a simplified pathway in which SQOR oxidizes H 2 S to generate glutathione persulfide (GSSH), which is then converted to sulfite and sulfatethus avoiding the need for sulfite to directly cross membranes (Landry et al., 2017)

Cancer Res 1977

The pain-relieving aspect appears secondary to tissue repair rather than direct analgesic action

I, II, III, IV and V located in the inner mitochondrial membrane [41]
