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Description
GPX2 overexpressing MOC1 tumors had a more suppressive tumor immune microenvironment and responded less favorably to anti-cytotoxic T-lymphocytes-associated protein 4 IC therapy in mice.Conclusion GPX2 overexpression represents a novel potentially targetable effector of immune escape in cold tumors.
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Finally, we outline a strategic framework to overcome mediator toxicity and ensure the generation of biocompatible fragments, providing a roadmap for innovative applications in cosmetics, food preservation, and environmental remediation.
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