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10.1042/bsr20190681 11 HuangX.LiangR.LiuY.YuL.YangM.ShangB.et al (2024)

Previous studies in keratinocytes indicated that adequate intracellular stores of GSH protect against peroxide-mediated loss of GPx-1 activity, as addition of GSH to cultures minimized enzymatic inactivation of GPx-1 by exposure to high concentrations of peroxides, whereas buthionine sulfoximine, an inhibitor of GSH biosynthesis, augmented sensitivity to peroxide-induced GPx-1 inactivation (373)

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Developing covalent protein drugs via proximity-enabled reactive therapeutics

Besides the aforementioned complex neuroendocrine interactions, a proper adrenal gland GC response during stress depends on the availability of cholesterol the major substrate for steroidogenesis [67], hereditary factors [68], and the crosstalk between multiple cell types coexisting in the adrenal microenvironment, such as adrenocortical, chromaffin, endothelial, neuronal, immune, and glial cells [69, 70]