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Therefore, we adopted LCA (150 mg/kgBW/d) to facilitate FAO, and further examined the AF vulnerability and atrial remodeling in vivo (a mice model of high-fat diet (HFD)-induced obesity-mediated AF) and in vitro (a primary atrial cardiomyocyte cell model of palmitate (PA)-mimicked lipid overloading), aiming to determine whether enhancing FAO can alter the process that underlie AF in obesity and explore possible mechanisms

432,438 Notably, the analgesic response to intravenous lidocaine, a sodium channel blocker, in PDPN patients correlates with increased SSC volume and enhanced functional connectivity between insular and corticolimbic circuits

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Camilo Fernndez Espina Colaboracin: Dr

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