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doi: 10.1242/dev.129304 29 LombardiMParolisiRScaroniFBonfantiEGualerziAGabrielliMet al

This signaling can further recruit additional inflammatory cells to the site of the identified pathogen, helping to clear the infection

H.JungH.JangJ

10.1038/s41419-025-07510-6 17 DixonS

Further research is needed to elucidate the complex interactions between ER stress, the UPR, and T cell immunity to develop novel therapeutic strategies for T cell-associated diseases
