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Early prediction of developing type 2 diabetes by plasma acylcarnitines: a population-based study
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The core argument is that estrogen acts as an endogenous, multi-target inhibitor of ferroptosis, conferring robust anti-ferroptotic capabilities to vascular cells by activating the Nrf2 central antioxidant system, optimizing cellular iron homeostasis (e.g., upregulating FPN1 and inhibiting ferritinophagy), and exerting direct antioxidant effects
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