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For example, a mutation in CACNA causes enhanced glutaminergic transmission because it allows the channels to open at more hyperpolarized membrane potentials than usual, which causes a more significant calcium influx to the terminal during the action potential

Guided by this structural prediction, we introduced cysteines into GT and UB2H domains (PBP1b E187C - R300C ) in the background of a functional truncation of PBP1b 40,41 encompassing residues 58-804 (Fig

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Defective protein secretion within Paneth cells has been linked to Crohns ileitis