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Description
This cell demise mechanism is fueled by iron and entails the degradation of cellular lipids, leading to significant oxidative damage and metabolic disturbances (Xiong et al., 2022
All studies are conducted in controlled laboratory environments

Its primary biological functions center on tissue remodeling: it stimulates collagen synthesis (types I and III), promotes decorin production, increases glycosaminoglycan synthesis, and activates metalloproteinase pathways for controlled tissue remodeling

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