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Keywords: Cuprizone, Multiple Sclerosis, Demyelination, Oligodendrocytes, Inflammation, Cell death, Astrocytes, Microglia, CNS Background Multiple Sclerosis (MS) is a chronic neurological disorder characterized by the loss of myelin or demyelination

Neuroactive peptides work through mechanisms such as brain-derived neurotrophic factor (BDNF) upregulation, HGF/c-Met signaling, and modulation of monoamine neurotransmitters (Table 4)

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Senolytics and senostatics: A two-pronged approach to target cellular senescence for delaying aging and age-related diseases