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This is probably a compensatory reaction against impaired ATP production due to the substrate-level glycolysis, and the same effect has been described in the ischemic/reperfused heart 21,22

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Mechanistically, these flavonoids increased dichlorofluorescein (DCF) oxidation, suggesting their ability to elevate intracellular ROS levels and activate p38 Mitogen-activated protein kinases (MAPKs)