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In a study that investigated the effect of BPC-157 on a rat model of hind limb ischemia, angiogenesis was promoted, helping to restore blood flow to the starved tissues by upregulating VEGFR2, a key regulator of angiogenesis [3]

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By donating its methyl group, SAM is converted first to S-adenosyl homocysteine (SAH) and then to homocysteine