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TSLP, IL-25, and IL-33 activate type 2 innate lymphoid cells (ILC2), which in turn produce Th2-associated cytokines such as IL-4, IL-5, and IL-13, thereby driving type 2 lung inflammation [30,31]

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This implies that the production of ROS alone does not determine lifespan to the same extent as the degree of oxidative damage sustained by cells
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