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Thalidomide: An Old Sedative-Hypnotic With Anticancer Activity

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In particular, central myelin degeneration promoted by pathological interactions involving both peripheral and central cell-mediated immune attacks induces the accumulation of dysfunctional myelin and neurological deficits 1,11

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Background: Pathogenic mutations in the LRRK2 gene are believed to contribute to PD pathogenesis via elevated LRRK2 kinase activity, thereby making LRRK2 kinase inhibition a promising therapeutic option