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This hypoxic environment leads to the stabilization of hypoxia-inducible transcription factors (HIFs), particularly HIF-1 and HIF-2, which regulate the transcription of many genes associated with endothelial cell proliferation and differentiation [via angiogenesis, such as vascular endothelial growth factor (VEGF)], extracellular matrix remodeling, and fibrogenesis [102]

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