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Description
D-lactic acidosis: turning sugar into acids in the gastrointestinal tract
The specific mechanism may be by activating VEGF-A / VEGFR1 -mediated AKT and p38/MAPK signaling pathways to upregulate downstream eNOS expression, which may increase the NO product synthesis thus promote angiogenesis of gastric mucosa, and also interact with ER stress

Stable gastric pentadecapeptide BPC 157 and NO-system

22 DaltonD
Non sono state osservate evidenze di effetti avversi ormonali, epatici o sistemici nei dati disponibili