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Collectively, our study identifies the L2HGDH-L2HG axis as a previously unrecognized metabolic checkpoint controlling ferroptosis sensitivity in liver cancer and uncovers glutathione degradation as a therapeutically exploitable vulnerability for ferroptosis-based treatment strategies in HCC.

This review examines butyrates origins, functions, and mechanisms in the gut, its impact on the gut-brain axis, and its relevance in both brain-first and gut-first PD models

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