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A specific mechanism by which glutathione thiol and glutathione disulfide operate to this aim is the reversible thiol(ate)-exchange mechanism, through which an excess of glutathione thiol regulates in an inhibitory sense the biological pathways which produce reducing cofactors or conditions by binding to sensing, conformation-determining disulfide bonds of proteins in a process termed reversible protein glutathionylation [134]

doi: 10.1111/j.1432-2277.2004.00005.x

10.1155/2016/7857186 46 HoughtonC

Since N- nitroso- N -methylurea was listed in the Second Annual Report on Carcinogens, additional studies in experimental animal have been identified that confirmed the induction of several tumor types previously observed and reported tumors at additional tissue sites, in additional species, and by additional routes of exposure: Tumors of the thymus were observed in rats following oral exposure (Lijinsky and Kovatch 1996)

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