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Given the exhaustion of GSH is a pivotal mechanism in ferroptosis, these findings support the notion that ferroptosis may be a key pathological process in intrinsic DILI

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doi: 10.1016/j.bbamcr.2023.119490

Together, these in vitro results suggest that lipid peroxidation, a surrogate of oxidative stress, can further exacerbate NLRP3-inflammasome activation by amplifying the induction of pro-IL-1 in response to SARS-CoV-2

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