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Ji W, Zhu Y, Li Y, Yang L, Zhao X, Cai H, et al
In one such study, Hotta and colleagues generated a non-obese diabetic (NOD, a T1D model) transgenic mouse in which human thioredoxin was over-expressed under control of the human insulin promoter (53)
This suggests that AMPK both promotes the nuclear translocation of MOTS-c and mediates its physiological effects, forming a positive feedback loop
These improvements correlate with histological findings of increased dermal thickness, enhanced collagen fiber organization, and reduced elastic fiber fragmentation
