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[DOI] [PubMed] [Google Scholar] 19.Zalups R.K., Ahmad S

Elevated ROS levels exert a paradoxical effect: while excessive ROS can be cytotoxic, moderate overproduction contributes to tumorigenesis by inflicting oxidative damage on DNA

Enzymes of the acyl-CoA thioesterase (ACOT) family, that are responsible for the deactivation of fatty acyl-CoAs, release the CoA and the esterified fatty acid

Plasma Glutathione Multiple processes control the plasma GSH/GSSG redox state, including GSH synthesis from its constitutive amino acids, cyclic oxidation and reduction involving GSH peroxidase and GSSG reductase, GSH transport into the plasma, and GSH and GSSG breakdown by -glutamyltranspeptidase [49,78,79]

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