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The principal aim of the treatment should be to improve bladder emptying and to reduce the risk of urinary tract infection
Inflammation stimulates the production of reactive oxygen species (ROS), which can further damage mitochondria and impair their function [103]
Therefore, we used 2-DG to inhibit glycolysis on top of the treatment with UK5099/Etomoxir/BPTES and found the OCR of T47D-LIP cells now decreased to a similar extent than control T47D cells ( Fig
