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Consistent with these findings, APC-mutant colonic organoids displayed selective sensitivity to Molidustat that was not reproduced by hydroxylase inhibition alone supporting a synthetic lethal interaction between GSTP1 and PHD2 in APC-mutant contexts
These were segregated into two coordinated clusters that distinguished the double knockout from all other genotypes (Fig
doi: 10.1007/s10552-009-9399-7
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