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This same study also found that exposing human airway epithelial cells (H292 cells) to electronic cigarette aerosols resulted in a dose-dependent increase in pro-inflammatory cytokines IL-8 and IL-6 (Lerner et al

When cells are exposed to certain death ligands or pathogen-associated molecular patterns (PAMPs), their response can escalate from pro-inflammatory and pro-survival gene expression to extrinsic apoptosis or programmed necrosis if there is an immediate threat, manifested as damage to key signaling components [4]

Nature 424, 938942

doi: 10.1097/med.0000000000000377 64 LiZMHernandez-MorenoDMainKMSkakkebaekNEKivirantaHToppariJet al

BPC-159 is a structurally different molecule, but without comparative studies, its impossible to say if its more effective, less effective, or simply different