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Depleted intracellular glutathione has been linked to Th1/Th2 immune imbalance and increased pro-inflammatory cytokine activity, which may contribute to autoimmune thyroid disease progression (Drge, 2002)

Researchers hypothesize this may influence: NAD metabolism methylation pathways cellular energy balance adipocyte metabolism Q12: Does it affect fat cells directly

Kadiyala N, Hovorka R, Boughton CK

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Antibody to -enolase has been identified in some patients (79) but this antibody is also frequently found in other autoimmune diseases