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In terms of mechanism, adiponectin can stimulate AMP-activated protein kinase (AMPK) phosphorylation and activation (43), increasing fatty acid oxidation and energy expenditure, partly through PPAR activation (44) in the liver and skeletal muscle

these changes included morphological alterations of mitochondria, iron accumulation, and oxidative stress [168]

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414 Amounts of products can be determined gravimetrically or by UV spectroscopy using = 24 000 at the max (around 278 nm)
84 :1509-18 Corresponding authors: Yunfei Ling, Associate Professor of Cardiovascular Surgery, Department of Cardiovascular Surgery, West China Hospital of Sichuan University, No
