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Inactivating mutations in the inwardly rectifying potassium channel 2.6 encoded by the KCNJ18 gene (in about 30%) have, among other candidates, been associated with susceptibility for developing thyrotoxic periodic paralysis (334, 335)

Stop and review with a clinician if symptoms persist

The hippocampus is particularly vulnerable to TBI, as evidenced in part by immediate mechanically induced damage to interneurons in the dentate granule cell layer of the hippocampus that are remote from the site of insult (Gallyas and Zoltay, 1992

and HGF) [23] or bone marrow stromal cells with the application of cyclosporine, minocycline, and methylprednisolone [24])