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TNF- and IL-1 are key inflammatory mediators of IDD, positively correlating with the severity and promoting ECMs catabolism by up-regulating proteolytic enzymes, whilst inhibiting the expression and synthesis of proteoglycan (Le Maitre et al., 2005

287 Several in vitro studies targeting these mechanisms have yielded promising results, 238,288 such as anti-DTP therapy targeting GPX4 has shown reductions in drug-resistant cancer cells, 8 and inhibition of the mTOR pathway reversed DTP cells formation

The impact on blood pressure also differed based on genetic type