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A.UrbanA.et al

It has been safely used for this purpose since the 1980s

Concurrently with the evolution and establishment of PGHS/PG-dependent mode of NSAID toxicity, a second school of opinions advocating the direct effect of NSAIDs on mitochondria followed by generation of cellular oxidative stress and apoptosis has also strengthened its foothold as an independent pathway of NSAID-induced cytopathology [24]

NAC and GSH have been shown to dramatically increase the killing of Staphylococcus aureus neutrophils in vitro