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Proposed model of the main biological effects, both positive and negative, due to the consumption and restriction of methionine (Martnez et al., 2017)

Overall the above reported evidence on the involvement of GSH and the redox environment are only some issues that deserve to be further investigated to comprehend the several aspects characterizing the possible crosstalk between redox reactions and modulation of ATG

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Loss of p53 promotes the interaction between DPP4 and nicotinamide adenine dinucleotide phosphate oxidase 1 (NOX1), leading to the formation of the NOX1-DPP4 complex and mediating plasma membrane lipid peroxidation and ferroptosis [40]