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Description
Reduction of lower motor neuron degeneration in wobbler mice by N-acetyl-L-cysteine
While these approaches remain important, a growing body of scientific research suggests that autism may also involve deeper biological mechanisms including mitochondrial dysfunction, chronic neuroinflammation, immune imbalance, and disruption of the gutbrain axis

[39] demonstrated that mitochondrial transplantation in A-treated SH-SY5Y cells increased mitochondrial membrane potential (MMP), ATP levels, and antioxidant enzyme activity, while reducing reactive oxygen species (ROS) and A aggregation [39]
Lang U, Kornemark M, Aubert ML, Paunier L, Sizonenko PC

As the oxidative burden on the mitochondria is lifted, these organelles can begin to shift away from inefficient anaerobic glycolysis (which produces the brain-fog-inducing lactate) and back toward efficient aerobic ATP production