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In vitro studies have demonstrated that BPC-157 can activate signaling pathways that promote fibroblast survival and growth, even under conditions of oxidative stress or hypoxia that often characterize the microenvironment of an injured tendon

Because healthy proliferating cells do not rely on sustained FOXO4p53 sequestration for survival, they are less affected in experimental models

This again underscores the diverse responses of patients to treatments

This has been reviewed in detail recently

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