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The mechanism by which it responds to signals from interorgan communication contributes to our deeper understanding of the complexity of metabolic diseases and provides insights into the development of new therapeutic strategies

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Physiological values of ROS are required to sustain the signaling pathways controlling crucial cellular processes, among which are inflammation, differentiation, proliferation, homeostasis, and apoptosis, whereas the generation of dysregulated ROS can amplify these same pathways, thus fostering disease phenotypes associated with ROS, as seen in type II diabetes, cancer, and atherosclerosis [19]