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Description
Major pathophysiological pathways in DILI Drugs may induce damage in the liver through multiple molecular mechanisms, including direct hepatic toxicity, as well as innate and adaptive immune response

Engagement of transferrin receptor by polymeric IgA1: Evidence for a positive feedback loop involving increased receptor expression and mesangial cell proliferation in IgA nephropathy

After screening and removing duplicate values, 3534 targets were identified (Fig
Report of the Multicenter Criteria Committee

Inference and analysis of cell-cell communication using CellChat
