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(2014, Nature Medicine) established NNMT as a major regulator of metabolic rate in white adipose tissue, showing that NNMT knockdown in mice produced a lean phenotype with elevated energy expenditure providing the foundational rationale for NNMT inhibition as a metabolic research target

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Knockout of ACSL4 in ferroptosis-sensitive murine and human cells conferred protection from erastin- and RSL3-induced cell death, while loss of ACSL4 predisposed cells to necroptosis [214]

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