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Multifactorial chylomicronaemia syndrome (MCS) has a prevalence of 1 in 600 of the population, resulting from a combination of heterozygous variants in the six FCS genes and/or accumulated common small-effect triglyceride-raising polymorphisms identified in genome-wide association studies (GWAS), such as APOA1-C3-A4-A5 , TRIB1 , LPL , MLXIPL , GCKR , FADS1-2-3 , NCAN , APOB , PLTP and ANGPTL3 [9, 10]

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