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The acute hemolytic attacks are the result of reduced antioxidant capacity of red blood cells with the result being membrane lipid peroxidation and consequent cell lysis

TGF- pathway activation occurs with upregulation of TGF- superfamily genes for tissue repair, while simultaneously suppressing pathological TGF-1 secretion in fibrotic conditions

First discovered in human plasma in the early 1970s, this molecule has since gained attention for its regulatory role in numerous biological pathways related to repair, regeneration, and protection

From a negative perspective [1], outside of the cytoprotection concept, or if cytoprotection is not a valid concept, these might indicate the uncertain complexity and multifaceted nature of the biological activity and its interaction with multiple systems