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Erectile dysfunction drugs altered the activities of antioxidant enzymes, oxidative stress and the protein expressions of some cytochrome P450 isozymes involved in the steroidogenesis of steroid hormones

Moreover, BA treatment protects against ATO-induced cardiotoxicity by suppressing oxidative stress, inflammatory factor expression, and by enhancing endogenous antioxidant systems through the inhibition of the TLR4/NF-B signaling pathway [254]

Several animal models have been used in this systematic review, including four pharmacological models and one genetic model

A significant difference was observed in all treatments as compared with control and also same results were identified between treatments (Figure 10B)
