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This antioxidant plays a role in reducing inflammatory stress in plasma and lymphocytes by directly reacting with reactive oxygen species (ROS)

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Importantly, overexpression of keap1a or knockdown of hmox1a partially rescued the blood clot formation, suggesting that the disrupted Keap1-Nrf2 signaling is responsible for the venous thrombosis-like phenotypes in smarca5 mutants

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