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Despite the versatility and diversity of the drugs used, the currently existing pharmacological methods for the treatment of IS are not effective enough and require further study of the molecular basis of ischemic damage, the development of new therapeutic agents, and approaches to identify potential neuroprotectors

Some studies show that treatment of such individuals with folic acid reduces seizures while others show the condition to be aggravated [30,31]

There are three key phenomena essential for the mechanism of necrosis: (1) the recruitment of RIPK1 to the TNFR1 by the adaptor protein TRADD, followed by the recruitment of TRAF2, another adaptor protein, which then associates with cIAP1/2, leading to a reduced caspase activation

Saponin components of Dioscorea Opposita are summarized in Table 8

doi: 10.3389/fcell.2017.00043