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There are abundant possibilities for bringing c-MET inhibitors to the clinic as a targeted therapeutic drugs fostering checks through metabolic signaling, tumor-microenvironment and immune modulations in HNSCC
On the contrary, depletion of IGF1R delayed the repair of radiation-induced DNA double strand breaks (Trojanek et al., 2003)
Skp2, the FoxO1 hunter
doi: 10.2174/1568026033452258
